The Effects of tDCS and Montoya Stair Task on Sensorimotor Recovery and GFAP Expression in MCAo induced Stroke Rat Model

The motor injury were took damage of the cerebral motor cortex, premotor cortex, motor pathway(motor tract) and related fibers, cerebrumandcerebellumpassage between the association pathways in the ischemic and/or hemorrhagic caused stroke(1). Especially, an experimental method that middle cerebral artery occlusion(MCAo) in the focal ischemic brain damage inducing a state of abnormal blood flow by artificially limit, and serumcalcium transient decrease. Therefore, inselectiveareasof theMCAsuppledbloodcortex, cells were death cause of this action. We has attention to supplies of blood flow specific area at middle cerebral artery that thebrainarea is involved in limbmovement and sensory motor functional recovery of upper limb. Theoccurred focal ischemicstroke is structuraldamage to the widespread connected brain that excitatory and/or inhibitory neurotransmitter deliver in each area(2, 3).This ismadeworsebysecondarybraindamagecausesseriouscomplicationsafterastroke. Recently, the protocol of non invasive trans-cranial direct current stimulation(tDCS) were known that stimulatewayofbrain regulates the functionof specific neural structures and the plasticity of the brain. Traditionally, the electrical stimulation to the brain through the nervous system in the rehabilitation process can improve the exercise training(4). The development of these brain stimulation techniques will possible to stimulate the non invasive and without pain.Also, stimulus intensity andduration, stimulation parameters such as various controlled to functional The Effects of tDCS and Montoya Stair Task on Sensorimotor Recovery and GFAP Expression in MCAo induced Stroke Rat Model